At the University of Arizona Medical Center, we are recruiting patients for two studies, among many others, that may have the potential to lead to improved healing of wounds and diabetic foot ulcers.
The first study involves a spray-on skin solution. The technology works in a similar way to bioengineered tissues. The big difference is that because it has a spray-on quality, the skin solution can go over a larger surface area and perhaps the contact with wounds and delivery may be better than previous iterations.
The product in the second study is an oligonucleotide, a gap junction modulator to reduce chronic inflammation in diabetic foot ulcers. Evidence seems to support that modulating gap junctions between cells and cell communication may modulate inflammation and also improve the rate of healing. This is looking very promising as well.